atherosclerosis造句
31. Methods The authors reviewed 42 patients underwent amputation for dry gangrene of leg due to atherosclerosis obliterans.
32. Disorders of lipid metabolism and atherosclerosis are common pathologic changes.
33. For hyperlipidemia, can be used for atherosclerosis, pressure, fatty liver of adjuvant therapy.
34. Progression of Subclinical Coronary Atherosclerosis: Does Obesity Make a Difference?
35. Changes of endothelial function and C - reaction protein ( CRP ) in hyperlipidemia and early atherosclerosis ( AS ).
36. AIM: To observe the effect of pravastatin on carotid artery atherosclerosis.
37. Objective To explore the clinical value of the abdominal aorta atherosclerosis spots observed with ultrasonography.
38. Background-The role of cholesteryl ester transfer protein in the development of atherosclerosis is still open to debate.
39. Measuring CIMT using ultrasonography is a well - accepted, noninvasive way to assess atherosclerosis.
40. And the typical atherosclerosis mottling in aorta lumen in model group under macrography and optical microscope were visible.
41. Patients with hyperlipidemia, hypercholesterolemia, atherosclerosis and other cardiovascular and liver diseasesmust eat carefully.
42. The abnormality of serum lipid is the major risk factor of atherosclerosis and CHD, and it also increases the susceptivity of hypertension, obesity and insuline resistance.
43. Clinical phases of pathological state and treatment based on differentiation of signs of coronary atherosclerosis.
44. This article reviews the evidence and mechanism that human cytomegalovirus causes atherosclerosis.
45. Endothelial cell apoptosis plays a key role in the development of atherosclerosis.
46. Conclusion Pravastatin can decrease blood lipid dramatically, relieve atherosclerosis, improve endothelium function and enhance the total treatment efficiency rate for angina.
47. The exact cause is unknown, but risks include high blood pressure, smoking, atherosclerosis (hardening of the arteries), trauma and Marfan's syndrome.
48. BackgroundPathologic studies in animals and human have clearly demonstrated that proliferation of adventitial vasa vasorum was associated with progression of coronary atherosclerosis.
49. Conclusion During the diagnosis of aortal atherosclerosis, the imaging velocity of B-TFE sequence is faster so that it is very valuable.
50. Atherosclerosis ( AS ) is a leading cause of cerebral infarction, especially the arterothrombtic cerebral infarction ( ACI ).
51. The creep curve is the relationship between changes in index, atherosclerosis of the ascending aorta has a certain impact on the creep characteristics.
52. Although the risk of losing a limb is low, hardening of the arteries in the legs is a marker for generalised atherosclerosis.
53. With sufficient funding, the focus on atherosclerosis would be broadened so that neurodegeneration, macular degeneration and lipofuscin accumulation would all also be targeted simultaneously.
54. Study on the Antioxidational Activity of Plasma on Experimental Atherosclerosis in Rabbits.
55. Recently, a preponderance of evidence supports thatinflammation plays an important role in a wide range of cardiovascular diseases, especially in the development of atherosclerosis.
56. Besides, diabetics , atherosclerosis patient , cholecystitis and pancreatitis patient do not drink the full-cream leben containing candy best , easy to aggravate state of illness otherwise.
57. The hemorheology indexes measured for 30 male patients with cerebral atherosclerosis in the paper.
58. Objective To investigate the utility of B-TFE sequence in the diagnosis of aortal atherosclerosis.
59. As one of proinflammatory cytokines, Interleukin - 18 ( IL 18 ) is highly expressed in atherosclerosis plaques.
60. Progression of symptomatic intracranial large artery atherosclerosis is associated with a proinflammatory state and impaired fibrinolysis.
12345